“I got the Shot and I’m fine...” Not so fast.
What is going to happen to the brains of babies?
The reports of deaths and injuries from the COVID shots continue to pour in. I feel compelled to write a few more articles, focusing on the EVIDENCE, in an attempt to stop the coming carnage to humanity, but particularly to children. They have no voice. They can’t refuse. Adults can voluntarily roll up their sleeves…or not. But who will stand in the gap for the fetus (pregnant women) and 3 to 6-month-old infants?
I’m going to take you through some rather heavy science about the blood-brain barrier and a group of proteins in the brain called tau proteins. Hang with me; this is important for understanding what’s coming.
Abstract: (simplified)
The blood-brain barrier (BBB) plays a crucial role in maintaining the specialized microenvironment of the central nervous system (CNS)...The list of CNS diseases associated with BBB dysfunction is growing. Opening the BBB, which will allow foreign particles in the blood to enter the brain, can lead to a host of abnormalities including chronic neuroinflammation, [cognitive] dysfunction, vascular dementia, strokes (hypoxic and ischemic), Alzheimer’s disease (AD), Parkinson’s disease, multiple sclerosis, amyotrophic lateral sclerosis (ALS), and even diabetes mellitus.
...BBB damage is also associated with conditions known as tauopathies (pronounced taw-op-a-thees). Tauopathies represent a group of approximately 20 different neurodegenerative diseases characterized by abnormal deposition of the tau proteins throughout the brain. This article summarizes the current understanding of the role of tau proteins when the BBB undergoes structural and functional changes.
REF: Michalicova, Alena, et al. “Tau Protein and Its Role in Blood-Brain Barrier Dysfunction. Frontiers in Molecular Neuroscience. 13:570045. Sept. 20, 2020. (full text) https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7554615/pdf/fnmol-13-570045.pdf
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The tau proteins are a group of six proteins. Their primary function is to maintain the stability of microtubules in the nerves within the brain and throughout the central nervous system (CNS). Found primarily in the brain, tau proteins are also found in lesser quantities in every cell in the body. They help to keep cellular microtubules strong, stabilize DNA, and regulate the transport of substances into neurons.
When tau proteins are damaged, they become misfolded and lose normal function. They clump together and form neurofibrillary tangles. Much has been published about how these tangled neurons can lead to brain pathologies. Neurofibriles damage what is referred to as the neurovascular unit (discussed below). Moreover, the distribution and amount of neurofibrillary tangles have been correlated with the severity of cognitive impairment seen within individuals.
When the tangled proteins spread throughout the brain, the disorders are called tauopathies. Tauopathies are characterized by either an accumulation of a pathological substance referred to as amyloid or by the release of fragments of tau proteins in the brain into the circulation. Clinical symptoms include frontotemporal dementia, akinesia with gait freezing, and cerebellar ataxia. Evidence exists that tau proteins may play a role in drug-resistant epilepsy, contributing to cognitive decline and continual neurodegeneration.
(diagram from Aaseth et al: https://www.mdpi.com/1660-4601/17/4/1269/htm)
Disruption of the BBB
The CNS is considered to be one of the most delicate systems in the human body. In fact, it has been estimated that almost every neuron has its own endothelial cells*. This fact explains the necessity to maintain a highly restrictive environment within the brain’s cells.
The blood-brain barrier (BBB) is a term used to describe the unique microvasculature of the central nervous system (CNS). The tight junctions of the BBB form a physical barrier that strictly regulates what is allowed to get into the brain, protecting it from toxins, pathogens, and chemical injury. The BBB can break down when it is exposed to chronic inflammation by pro-inflammatory cytokines or chemokines, or by exposure to chemical and environmental toxicities.
Previously, the BBB was characterized as a single layer of endothelial cells*, a monolayer of cells connected to one another by what is referred to as the ‘tight junctions.’
*DEFINITION: Endothelial cells form the barrier between vessels and tissues. They control the flow of substances and fluid into and out of a tissue. Endothelial cells line blood vessels and lymphatic vessels. Impaired function can lead to serious health issues within the brain.
More recently, the BBB has been found to be part of what is called the neurovascular unit (NVU). The NVU is a highly dynamic system that oversees the proper functioning of the brain. The NVU is interdependent with the function and interaction between neurons, mast cells, glial cells, immune cells, and even tissues outside the brain in the peripheral body. This ‘intelligent network’ can influence processes throughout the entire nervous system. An intact BBB is crucial for the proper function of the NVU.
The breakdown of the BBB can be caused by physical disruption of the tight junctions (such as a concussion or head trauma) and/or enzymatic degradation of the basement membranes of the endothelial cells. The list of CNS pathologies involving BBB dysfunction is rapidly expanding. BBB disruption has now been associated with numerous pathological conditions such as ischemia, infections, epilepsy, tumors, and neuroinflammatory diseases including tauopathies. The breakdown of the BBB is an early, independent biomarker of human cognitive dysfunction.
It has recently been confirmed that tau proteins can cross into the brain or out of the brain into the blood . When the BBB is damaged, tau proteins can leak into the blood. In fact, tau proteins have been found in the blood of 40 - 50% of patients in the acute phase of a stroke when the BBB is disrupted.
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